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Gustavo Duque, MD, PhD, is an Assistant Professor of Medicine
and Geriatric Medicine. He is the Coordinator of Undergraduate
Medical Education at the Division of Geriatric Medicine. He
is also a Teaching Scholar at McGill University and a Macy
Scholar in Medical Education at Harvard University . His education
interests relate to the education of Aging and Geriatrics
throughout the medical curriculum. His main projects include
the Trans-Curricular Teaching of Aging and Geriatrics (T.T.A.G.
Project) and the McGill Electronic Evaluation Portfolio (MEEP).
In addition, he is a clinician at the Division of Geriatric
Medicine, Jewish General Hospital, and a researcher at the
Bloomfield Centre for Aging Studies at the Lady Davis Institute
for Medical research.
CLICK HERE TO VISIT HIS WEBSITE
UNIVERSITY POSITIONS
Assistant Professor, Department of Medicine & Division
of Geriatric Medicine - McGill University
Member - Bloomfield
Centre for Studies in Aging
Co-Coordinator Undergraduate Medical
Education - Division of Geriatric Medicine
Teaching Scholar -
McGill University
Associated Member - Centre for Medical Education,
McGill University
HOSPITAL APPOINTMENTS
Physician, Division of Geriatric Medicine, Department of
Medicine
Sir Mortimer B. Davis-Jewish General Hospital , McGill
University
PUBLICATIONS
1. |
Chang G, Duque G.2002.
Age-related bone loss: old term new facts. Gerontology
48:62-71. MEDLINE link to this publication |
2. |
Duque G , El Abdaimi K, Macoritto M,
Miller, M. M. Kremer R. Estrogens (E2) regulate expression
and response of 1,25-dihydroxyvitamin D3 receptors in bone
cells: changes with aging and hormone deprivation. 2002.
Biochem Biophys Res Commun. 299(3):446-54 MEDLINE link to this publication |
3. |
Duque G. Will reducing
adipogenesis in bone increase bone mass? : PPAR gamma
2 as a key target in the treatment of age related bone
loss. 2003. Drugs News & Development.
16:1-6. MEDLINE link to this publication |
4. |
Duque G , Macoritto M, Kremer R. 1,25(OH)
2D 3 inhibits bone marrow adipogenesis in senescence accelerated
mice (SAMP-6) by decreasing the expression of peroxisome
proliferators-activated receptor gamma 2 (PPAR g 2). 2004.
Experimental Gerontology, 39: 333-338. MEDLINE link to this publication |
5. |
Duque G, El-Abdaimi K, Henderson
JE, Lomri A, Kremer R. Vitamin D exerts its
anti-apoptotic effect in osteoblasts through a Fas related
mechanism. 2004. Bone, 35:57-64. MEDLINE link to this publication |
6. |
Duque G , Macoritto M, Kremer R. Vitamin
D treatment of senescence accelerated mice (SAM-P/6) induces
several regulators of stromal cell plasticity. Biogerontology
5(6):421-9. MEDLINE link to this publication |
7. |
Duque G , Dion N, Macoritto M, Ste.
Marie LG, Kremer R. 1,25(OH) 2D3 acts as a bone forming
agent in the hormone independent Senescence Accelerated
Mouse (SAMP6) model of Osteoporosis. The American Journal
of Physiology: Endocrinology and Metabolism 2005 Apr;288(4):E723-30.
2004 Nov 30. [Epub ahead of print] MEDLINE link to this publication |
8. |
Joseph C, Kenny A, Lorenzo, Duque G and
Kuchel G. Role of Endocrine-Immune Dysregulation in Osteoporosis,
Sarcopenia, Frailty and Fracture Risk. Molecular Aspects
of Medicine 2005 ;26(3):181-201 MEDLINE link to this publication |
9. |
Montero-Odasso M, Duque G, Vitamin
D in the aging musculoskeletal system: an authentic strength
preserving hormone. Molecular Aspects of Medicine 2005
;26(3):203-19 MEDLINE link to this publication |
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